By Muriel Chiron-Charrier
Marie Curieuse... mais qui est donc Marie Curieuse ? Ce pourrait être vous, si tout comme elle, des questions vous assaillent dès le matin à los angeles sortie du lit : Quelle différence y-a-t-il entre l. a. théine et los angeles caféine ? remark le savon a-t-il été créé ? A quoi sert le collagène présent dans notre épiderme ? Avec quoi se maquillait Cléopâtre ?
Sur un ton à los angeles fois drôle et sérieux, l'auteure nous fait découvrir l'histoire d'un produit, les origines de ses composants, les réactions chimiques pour arriver à un tel résultat et son utilisation... bref l. a. petite chimie du matin ! Chaque partie est construite autour d'une thématique.
Les explications sont claires et concises et les illustrations décalées nous emportent dans le monde de Marie Curieuse.
Read or Download Café, crème, savon et Cie: La petite chimie du matin de Marie Curieuse PDF
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Additional info for Café, crème, savon et Cie: La petite chimie du matin de Marie Curieuse
Example text
Several studies have reported that HIF-2α is upregulated in tumor parenchymal cells and in tumor-associated macrophages (TAM), which are recruited to the hypoxic, avascular regions of tumors by growth factors and chemokines resulting in tumor progression [81,85,86]. A number of HIF-regulated angiogenic factors, such as vascular endothelial growth factor (VEGF), basic fibroblast growth factor (bFGF), VEGF receptor (VEGFR), IL-8, iNOS, and angiopoietins, are released by macrophages [81,87]. Many of these factors further accelerate inflammatory angiogenic process, thereby triggering tumor growth [88].
Indd 8 8/21/2008 3:23:27 PM Signaling Molecules as Targets of Dietary Chemopreventives 9 abnormally proliferating cells that comprise premalignant or malignant tumors by the modulation of cell cycle progression is another important strategy for chemoprevention as well as therapy [4,66]. A cell cycle consists of four different phases: gap I (G1), synthesis (S), gap 2 (G2), and mitosis (M). During the G1 phase, cells grow in size and get prepared for DNA replication. The S phase involves the replication of chromosomes, and the G2 phase checks the completion of DNA replication and prepares cells to undergo mitosis.
Capsaicin induced apoptosis in HL-60 cells, human esophagus epidermoid carcinoma cells, and B16-F10 melanoma cells, which was accompanied by the decrease in levels of mitochondrial membrane potential, enhanced cytochrome c release, and increased activation of caspase-3 [255,257,258]. Treatment of HT-29 cells with capsaicin caused apoptosis via upregulation of AMPK [259]. Capsaicin caused G1 arrest in endothelial cells that was correlated with the downregulation of the expression of cyclin D1-Cdk-4 and reduced phosphorylation of Rb protein.
