By Professor Michael Forsting MD, PhD (auth.), Michael Forsting MD, PhD, Isabel Wanke MD, PhD (eds.)
This booklet describes the pathoanatomical, pathophysiological, and imaging positive factors of vascular malformations and aneurysms of the mind and the fashionable, minimally invasive endovascular equipment and methods hired of their remedy. person chapters are dedicated to developmental venous malformations, cavernomas and capillary telangiectasias, pial arteriovenous malformations, dural arteriovenous malformations, and intracranial aneurysms. every one bankruptcy is subdivided into 4 important sections on pathology, scientific presentation, diagnostic imaging, and treatment, making sure a standardized technique all through. All chapters during this 2d revised version of Intracranial Vascular Malformations and Aneurysms were completely up-to-date. The ebook is richly illustrated with quite a few informative CT, MR and DSA pictures, together with high-end 7-Tesla MR images.
Read Online or Download Intracranial Vascular Malformations and Aneurysms: From Diagnostic Work-Up to Endovascular Therapy PDF
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Extra info for Intracranial Vascular Malformations and Aneurysms: From Diagnostic Work-Up to Endovascular Therapy
Example text
2001) had only minor complaints with disputable cause due to capillary telangiectasias, the patient reported by Huddle et al. (1999) had severe neurologic symptoms and died presumably due to the ensuing brainstem dysfunction. Both severely symptomatic patients showed an extensive T2 signal abnormality of the affected parts of the brainstem. Furthermore, for us there might be an association of tinnitus and pontine capillary telangiectasias. Further observations will be necessary to establish whether there is in fact a severely symptomatic, aggressive subform of capillary telangiectasias.
1999); the majority of studies, however, did not find any gender difference in bleeding risks. Other authors figured out that the most significant predictor of clinical events is lesion location. 7% for supratentorial lesions. Following another classification (deep vs superficial) deep lesions do have a significant higher bleeding incidence. Intuitively, the most plausible explanation for that is that the eloquence of deep structures will lead to clinical symptoms even with a small change in size of the lesion.
Hemosiderin or ferritin is a well-known epileptogenic agent (at least in animal experiments). Being aware of the relation between seizures and hemosiderin deposition is of particular importance if surgical removal of the cavernoma is considered due to conservative untreatable seizures. It is of utmost importance not only to remove those parts of the cavernoma with Cavernomas and Capillary Telangiectasias obvious cavernoma, but also to remove the hemosiderin deposits around the cavernoma within the adjacent brain tissue (Baumann et al.
