By Andrea Natale, Antonio Raviele
- Describes most sensible practices in Ventricular Tachycardia and traumatic inflammation Ablation in accordance with a consensus of the world's top specialists, in response to the once a year 'Venice Arrythmias' meeting.
- Catheter ablation of atrial traumatic inflammation has turn into a common, but controversial, therapy modality in electrophysiology laboratories.
- Presents a entire and up to date review of all of the most vital and debated facets of atrial traumatic inflammation ablation.
- Covers all key issues, from pre- and intra-procedural administration to top strategies and results for sufferers with and with no structural HD to symptoms for VT/VF ablation and hybrid remedy to destiny instruments and therapy options.
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Additional info for Ventricular Tachycardia Fibrillation Ablation: The state of the Art based on the VeniceChart International Consensus Document
Sample text
It is unclear if these racial disparities are the result of genetic differences, differences in the access to care, or differences in the environment. SCD has also been linked to cigarette smoking, obesity, and significant psychological stress [23,31–34]. Trends over time Over the past several decades, there has been a clear decrease in the incidence of ventricular arrhythmias causing SCD [35–37]. Data from the Framingham Study reveal that the age-adjusted risk of SCD has decreased by 49% during 1990–1999 as compared to the years 1950–1969 [35].
RVOT) or repetitive runs of VT [43,46]. Reentry requires (1) two or more potential pathways of activation; (2) unidirectional conduction block in one (or more); (3) slow conduction to allow the initial site of block time to recover and be re-excited to complete a reentrant circuit. Unidirectional conduction block may occur after a properly timed extra-beat or enhanced sinus rate, and is probably functional rather than fixed in most instances [47,48]. However, regions of conduction block can be anatomically fixed such that they are present during tachycardia and sinus rhythm; dense, non-excitable fibrosis or valve annuli create these types of anatomic boundaries for reentry [49].
Coronary artery anomalies with an abnormal origin in the sinus of Valsalva are found in 17% of the patients, and myocarditis and ARVC are found in 6% and 4%, respectively [88]. Differentiation of HCM from the so-called athlete’s heart is not an easy task [89,90]. This issue is to some extent speculative and it is beyond the scope of this review to further elaborate on this matter. When simple genetic diagnostic tests would be readily available, it would be possible to not only delineate the boundaries of the athlete’s heart but also establish which benign varieties of HCM might be allowed for competitive sports practice at no risk of SCD on the pitch.
